- Systems Neurobiology (BIO 340/440), Winter Quarter
- Topics in Neurobiology (BIO 341/441), Spring Quarter
- Biostatistics (BIO 206)
- Introduction to Pharmacology (BIO 375/475)
- Introduction to Graduate Studies (BIO 495)
- The Use of Animals in Science (ISP 101)
- Cognitive Neuroscience (BIO 342) Spring Quarter
My lab at DePaul University is focused on Traumatic Brain Injury (TBI) in a rodent model. We have examined numerous approaches aimed at repairing the injured brain following TBI including: gene therapy, bone marrow stem cell transplants, endogenous neurogenesis, and physical rehabilitation. Our current emphasis is the examination of neural plasticity following TBI and how it can be enhanced by physical rehabilitation in an animal model. We utilize steretotaxic surgery, quantitative anatomical analysis, and sophisticated behavioral techniques in our studies. Our overall goal is to understand how the brain tries to repair itself, and how external therapies can help the brain do so.
Kozlowski, D.A., Leasure, J.L., Schallert, T. (2013) The Control of Movement Following Traumatic Brain Injury.
Comprehensive Physiology. 3(1) 121-139. doi: 10.1002/cphy.c110005
D.A. & Jones, T.A. (2012) Plasticity and Recovery of the Injured Brain.
In. (Morganti-Kossman, C., Raghupathi, R., & Maas, A. Editors) “Traumatic brain and
spinal cord injury: Challenges and developments in research: Translating Research into Clinical Practice”.
Cambridge University Press.
Jones, T.A., *Liput, D.J.,
Maresh, E., Donlan, N. Parikh, T.,
Marlowe, D., Jones, T.A. & Kozlowski, D.A. (2012) Use-dependent
dendritic regrowth is limited following unilateral controlled cortical
impact to the forelimb sensorimotor cortex.
Journal of Neurotrauma
29(7):1455-1468. Article chosen to highlight for cover photo. *both authors contributed equally to
Yamada, K.H., *Kozlowski, D.A., *Seidl, S.E., Lance, S.,
Wieschhaus, A.J., Sundivakkam, P., Tiruppathi, C., Chishti, I., Herman, I.R., Kuchay, S.M., & Chishti, A.H. (2012).
Targeted gene inactivation of calpain-1 suppresses cortical
degeneration due to traumatic brain injury and neuronal apoptosis
induced by oxidative stress.
Journal of Biological Chemistry.
287(16):13182-13193. *all authors contributed equally to this manuscript
Professional Society Membership
- Society for Neuroscience
- National Neurotrauma Society
- Chicago Society for Neuroscience
- Faculty for Undergraduate Neuroscience